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  • The Anti-Obesity Potential of Green Tea: The Effect on Leptin and Adiponectin

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    EssexPUB3273.pdf (444.0Kb)
    Author(s)
    Essex, Kenneth
    Mosawy, Sapha
    Griffith University Author(s)
    Essex, Kenneth T.
    Mosawy, Sapha
    Year published
    2016
    Metadata
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    Abstract
    Green tea has been consumed for thousands of years, and has been known to have many medicinal properties such as, anti-cancerous, anti-atherogenic, anti-oxidative, anti-inflammatory and anti-bacterial. With the increasing emergence of cardiovascular disease (CVD), and its major risk factor, obesity, there has been increasing interest in green tea as a potential anti-obesity therapy. Evidence has demonstrated that green tea decreases body weight. However it remains unclear through which pathway and mechanism green tea exerts its anti-obesity properties. The metabolic adipose tissue hormones, adipocytokines, leptin and adiponectin ...
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    Green tea has been consumed for thousands of years, and has been known to have many medicinal properties such as, anti-cancerous, anti-atherogenic, anti-oxidative, anti-inflammatory and anti-bacterial. With the increasing emergence of cardiovascular disease (CVD), and its major risk factor, obesity, there has been increasing interest in green tea as a potential anti-obesity therapy. Evidence has demonstrated that green tea decreases body weight. However it remains unclear through which pathway and mechanism green tea exerts its anti-obesity properties. The metabolic adipose tissue hormones, adipocytokines, leptin and adiponectin have been studied extensively as a potential target for green tea and its main catechin, epigallacatechin gallate (EGCG). It has been demonstrated that green tea consumption decreases leptin levels which may directly decrease body weight through increasing energy expenditure and fat oxidation. Furthermore, ingestion of green tea or its catechins has been associated with increased adiponectin levels which may indirectly reduce body weight through insulin sensitivity.
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    Journal Title
    Clinical Immunology, Endocrine & Metabolic Drugs
    DOI
    https://doi.org/10.2174/2212707004666161228142812
    Copyright Statement
    © 2016 Bentham Science Publishers. This is the author-manuscript version of this paper. Reproduced in accordance with the copyright policy of the publisher. Please refer to the journal website for access to the definitive, published version.
    Subject
    Medical Biochemistry: Lipids
    Publication URI
    http://hdl.handle.net/10072/337432
    Collection
    • Journal articles

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