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dc.contributor.authorSahni, Sumit
dc.contributor.authorKovacevic, Zaklina
dc.contributor.authorKalinowski, Danuta S
dc.contributor.authorHuang, Michael
dc.contributor.authorMenezes, Sharleen
dc.contributor.authorKrishan, Sukriti
dc.contributor.authorLane, Darius JR
dc.contributor.authorJansson, Patric J
dc.contributor.authorRichardson, Des R
dc.description.abstractThe tumor microenvironment has received considerable attention in terms of designing pharmacological strategies for optimising cancer treatment. Considering this, it is notable that the microenvironment of the cancer induces cell survival pathways, for example, autophagy, to ensure neoplastic cells have access to energy and metabolites for growth and progression. Cancer cells are known to have increased autophagy, and a recent study has suggested that autophagy can promote metastasis. Thus, the identification of new drug targets to suppress autophagy can offer a unique strategy for novel drug development initiatives against aggressive and metastatic cancers.
dc.publisherFuture Science Ltd
dc.relation.ispartofjournalFuture Medicinal Chemistry
dc.subject.fieldofresearchMedicinal and biomolecular chemistry
dc.subject.fieldofresearchPharmacology and pharmaceutical sciences
dc.subject.keywordsScience & Technology
dc.subject.keywordsLife Sciences & Biomedicine
dc.subject.keywordsChemistry, Medicinal
dc.subject.keywordsPharmacology & Pharmacy
dc.titleTargeting autophagy in antitumor agent design: Furthering the 'lysosomal love' strategy (Editorial)
dc.typeJournal article
dc.type.descriptionC2 - Articles (Other)
dcterms.bibliographicCitationSahni, S; Kovacevic, Z; Kalinowski, DS; Huang, M; Menezes, S; Krishan, S; Lane, DJR; Jansson, PJ; Richardson, DR, Targeting autophagy in antitumor agent design: Furthering the 'lysosomal love' strategy (Editorial), Future Medicinal Chemistry, 2016, 8 (7), pp. 727-729
gro.hasfulltextNo Full Text
gro.griffith.authorRichardson, Des R.

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