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  • Chronic Fatigue Syndrome/Myalgic Encephalomyelitis and the Potential Role of T Cells

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    100150_1.pdf (999.8Kb)
    Author(s)
    Hardcastle, Sharni
    Brenu, Ekua
    Staines, Don
    Marshall-Gradisnik, Sonya
    Griffith University Author(s)
    Staines, Don R.
    Hardcastle, Sharni L.
    Marshall-Gradisnik, Sonya M.
    Brenu, Ekua
    Year published
    2014
    Metadata
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    Abstract
    Chronic Fatigue Syndrome/Myalgic Encephalomyelitis (CFS/ME) is a multifactorial disorder defined by symptom-specific criteria and characterised by severe and prolonged fatigue. CFS/ME typically affects a variety of bodily systems, including the immune system. Patients with CFS/ME exhibit significantly reduced Natural Killer (NK) cell activity suggesting immune which may be hallmarks of changes in the adaptive immune system, potentially including T cell subsets and function. The principal purpose of T cells is to regulate immune responses and maintain immune homeostasis. These regulatory measures can often be compromised ...
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    Chronic Fatigue Syndrome/Myalgic Encephalomyelitis (CFS/ME) is a multifactorial disorder defined by symptom-specific criteria and characterised by severe and prolonged fatigue. CFS/ME typically affects a variety of bodily systems, including the immune system. Patients with CFS/ME exhibit significantly reduced Natural Killer (NK) cell activity suggesting immune which may be hallmarks of changes in the adaptive immune system, potentially including T cell subsets and function. The principal purpose of T cells is to regulate immune responses and maintain immune homeostasis. These regulatory measures can often be compromised during illness and may present in a number of diseases including CFS/ME. This review paper examines the role of T cells in CFS/ME and the potential impact of T cells on CFS/ME immune profiles with an evaluation of the current literature.
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    Journal Title
    Biological Markers and Guided Therapy
    Volume
    1
    Issue
    1
    DOI
    https://doi.org/10.12988/bmgt.2014.3122
    Copyright Statement
    © 2014 S. L. Hardcastle et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
    Subject
    Cellular Immunology
    Publication URI
    http://hdl.handle.net/10072/69289
    Collection
    • Journal articles

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